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the sycosis parasitaria of authors, and it is probable he no
longer denies its existence.
Tilbury Fox (" Skin-Diseases," 2d American edition,
New York, 1873, p. 502) calls it a parenchymatous inflamma-
tion of the hair-follicles, with the ordinary results of long-con-
tinued congestion. He says it is an independent morbid state
of the actual hair-follicle itself, the cause of which is doubt-
ful, though external irritants, as shaving and long-continued
exposure to heat-rays, can produce it in persons "out Purchase Geodon of
health."
Nelligan ("Diseases of the Skin," 4th American edition,
Philadelphia, 1864, p. 275) considered all cases of sycosis as
parasitic.
Wilson (" Lectures on Dermatology," London, 1871, p. 136)
says it Buy Geodon Online is an inflammation of the hair-follicles. There are sev- Cheap Geodon
eral varieties of the Buy Geodon disease, and among them is observed the
phytiform degeneration of tlie epithelium consequent on de-
ranged development and nutrition of the tissue.
Hutchinson (" London Hospital Reports," vol. iii., 1866, p.
389) denies the existence of a parasitic sycosis.
McCall Anderson (" Eczema," 3d ed., London, 1814, cap.
xii.) describes under the term eczema pilare facicei appear-
ances which correspond exactly with those of sycosis; yet he
says it differs from the latter in being simply an extension
of the eczema to the perifollicular region, while sycosis con-
sists in small abscesses and tubercles.
Baudet (" Traite des Affections de la Peau," Paris, 1869,
p. 94) says it is an inflammation of the hair-follicles depending
on an arthritic constitution. He believes that a sycotic erup-
tion, whether arthritic or parasitic, can be converted into a
syphilitic or a scrofulous eruption if the patient, while afflected
with sycosis, becomes syphilitic or scrofulous.
The other French writers, as Cliauslt, Rayer, Duchenne,
Dupare, Gibert, Rochard, Divergie, Giubout, etc., have con-
tributed nothing worthy of special mention.
Wertheim {Zeitschrift der h. h. G^sellschaft der Aerzte,
1861) made use of the microscope in studying this disease,
but only with the object of confirming previously-conceived
ideas of its nature and cause. He argued thus: Since a
\
SYCOSIS. 127
hair is always in the centre of every pustule, and pus always
follows the extraction of such a hair, therefore the hair-folli-
cle must be the seat of the inflammation. Every such follicle
will therefore inclose an abscess with its sheaths, and ever^'^
pustule is a metamorphosed hair-follicle. As sycosis appears
but very rarely on other parts of the body than that of the
bearded parts of the face, therefore the cause of the disease
must be in the anatomical character of the hair-follicles of this
region. Believing in those conjectures, he examined the
structure of healthy hair-follicles taken from different parts of
the body. From these studies he found that in the beard the
hair has a greater diameter, in comparison with the diameter
of its sheaths, than in any other part of the body. As the pus
reaches the surface by destroying or pushing aside the exter-
nal and internal root-sheaths, therefore, when the hair-follicle
is inflamed, the pus has much more difiiculty in reaching the
surface when the hair is thick and the sheaths thin, than when
the hair is thin and its compressible sheaths thick. On account
of the resistance which a thick hair offers to the immediate
evacuation of pus, the latter collects, and an abscess is formed.
Kobner (" Klinische und experimentelle Mittheilungen
aus der Dermatologie und Syphilidalogie," Erlangen, 1864,
p. 13) was the first to give any description of the changes
which take place in the hair in sycosis. He names the dis-
ease folliculitis barbae, and says it has no connection with
acne, but is an idiopathic inflammation of the follicle, which
inflammation is the cause of the nutrition changes which take
place in the hair. This inflammation may be produced by
changes of temperature as well as from chemical and mechan-
ical irritants. lie examined hairs extracted in different
stages of the eruption, but did not make any microscopical
examination of the diseased follicles in situ or otherwise. The
changes outside the follicle he regards as consecutive to those
taking place within it. He says that first the external and
then the internal root-sheaths swell, their cells become en-
larged, increase in number, divide, and finally are partly
transformed into pus and detritus. The hair itself is not
changed at first, but later it loses its connection with the fol-
licle, the bulbi swell two or three times their normal size, and
128 THE STRUMOUS ELEMEJ!fT IN THE
become infiltrated with a sero-purulent fluid. According to
Kobner, the primary changes therefore take place in the ex-
ternal and internal root-slieaths. The cause of the perifol-
licular inflammation, he finds, lies in the anatomical construc-
tion of the part, in its richness in blood-vessels and nerves,
and the deep situation of the follicles.
Since the time of Bateman, therefore, the disease has been
regarded by almost all writers as an inflammation of the hair-
follicles, produced by irritating agents. Wertheim and Kob-
ner were the only writers who studied the disease with the
aid of the microscope, but they examined only hairs extracted
from the part affected, and not the whole tissue affected ; i. e.,
they did not examine the affected part itself.
In my own studies upon this subject I examined portions
of skin taken from the living subject and affected with the
eruption in its different stages, and have followed the process
from its commencement to its termination, either in complete
restitution of the parts, or in greater or less destruction of the
skin of the affected region.
{To he concluded^ with Illustrations, in next Number.)
Art. II. — The Strumous Element in the Etiology of Joint-
Disease, from an Analysis of Eight Hundred Order Geodon and Sixty
Cases. Bj V. P. Gebney, M. D., Assistant Surgeon to
the Hospital for the Ruptured and Crippled, New York.
{Concluded from July Number.)
This narrows down to two cases with a presumable ahso-
lutehj clear record. One of these, on admission, from the
history during her stay as in-patient, and under observation
subsequently as an out-patient, induced my predecessor in
the hospital. Dr. IT. E. Ilanderson, a very competent observer
and thoroughly intelligent physician, to gravely doubt the
existence of any hip-disease at all, and such doubts are ex-
pressed on his notes. I myself had an opportunity of observ-
ing the case, and saw no positive evidence of the disease in
question. The diagnosis was coxo-femoral neuralgia.
The remaining case was such a typical one of acute syno-
vitis of the coxo-femoral articulation, that in my summary I
ETIOLOGY OF JOINT-DISEASE.
129

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